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Dr. Simeons’ Original Protocol  
Pounds & Inches
  
A NEW APPROACH TO OBESITY
 
 
BY: A.T.W. SIMEONS, M.D. 
SALVATOR MUNDI INTERNATIONAL HOSPITAL 
00152 – ROME 
VIALE MURA GIANICOLENSI, 77 
FOREWORD 
This book discusses a new interpretation of the nature of obesity, and while it does not 
advocate yet another fancy slimming diet it does describe a method of treatment which has 
grown out of theoretical considerations based on clinical observation. 
What I have to say is, in essence, the views distilled out of forty years of grappling with 
the fundamental problems of obesity, its causes, its symptoms, and its very nature. In these 
many years of specialized work, thousands of cases have passed through my hands and 
were carefully studied. Every new theory, every new method, every promising lead was 
considered, experimentally screened and critically evaluated as soon as it became known. 
But invariably the results were disappointing and lacking in uniformity. 
I felt that we were merely nibbling at the fringe of a great problem, as, indeed, do most 
serious students of overweight. We have grown pretty sure that the tendency to 
accumulate abnormal fat is a very definite metabolic disorder, much as is, for instance, 
diabetes. Yet the localization and the nature of this disorder remained a mystery. Every 
new approach seemed to lead into a blind alley, and though patients were told that they are 
fat because they eat too much, we believed that this is neither the whole truth nor the last 
word in the matter. 
Refusing to be side-tracked by an all too facile interpretation of obesity, I have always 
held that overeating is the result of the disorder, not its cause, and that we can make little 
headway until we can build for ourselves some sort of theoretical structure with which to 
explain the condition. Whether such a structure represents the truth is not important at this 
moment. What it must do is to give us an intellectually satisfying interpretation of what is 
happening in the obese body. It must also be able to withstand the onslaught of all hitherto 
known clinical facts and furnish a hard background against which the results of treatment 
can be accurately assessed. 
To me this requirement seems basic, and it has always been the center of my interest. In 
dealing with obese patients it became a habit to register and order every clinical 
experience as if it were an odd looking piece of a jig-saw puzzle. And then, as in a jig saw 
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puzzle, little clusters of fragments began to form, though they seemed to fit in nowhere. 
As the years passed these clusters grew bigger and started to amalgamate until, about 
sixteen years ago, a complete picture became dimly discernible. This picture was, and still 
is, dotted with gaps for which I cannot find the pieces, but I do now feel that a theoretical 
structure is visible as a whole. 
With mounting experience, more and more facts seemed to fit snugly into the new 
framework, and then, when a treatment based on such speculations showed consistently 
satisfactory results, I was sure that some practical advance had been made, regardless of 
whether the theoretical interpretation of these results is correct or not. 
The clinical results of the new treatment have been published in scientific journal and 
these reports have been generally well received by the profession, but the very nature of a 
scientific article does not permit the full presentation of new theoretical concepts nor is 
there room to discuss the finer points of technique and the reasons for observing them. 
During the 16 years that have elapsed since I first published my findings, I have had many 
hundreds of inquiries from research institutes, doctors and patients. Hitherto I could only 
refer those interested to my scientific papers, though I realized that these did not contain 
sufficient information to enable doctors to conduct the new treatment satisfactorily. Those 
who tried were obliged to gain their own experience through the many trials and errors 
which I have long since overcome. 
Doctors from all over the world have come to Italy to study the method, first hand in my 
clinic in the Salvator Mutidi International Hospital in Rome. For some of them the time 
they could spare has been too short to get a full grasp of the technique, and in any case the 
number of those whom I have been able to meet personally is small compared with the 
many requests for further detailed information which keep coming in. I have tried to keep 
up with these demands by correspondence, but the volume of this work has become 
unmanageable and that is one excuse for writing this book. 
In dealing with a disorder in which the patient must take an active part in the treatment, it 
is, I believe, essential that he or she have an understanding of what is being done and why. 
Only then can there be intelligent cooperation between physician and patient. In order to 
avoid writing two books, one for the physician and another for the patient - a prospect 
which would probably have resulted in no book at all - I have tried to meet the 
requirements of both in a single book. This is a rather difficult enterprise in which I may 
not have succeeded. The expert will grumble about long-windedness while the lay-reader 
may occasionally have to look up an unfamiliar word in the glossary provided for him. 
To make the text more readable I shall be unashamedly authoritative and avoid all the 
hedging and tentativeness with which it is customarily to express new scientific concepts 
grown out of clinical experience and not as yet confirmed by clear-cut laboratory 
experiments. Thus, when I make what reads like a factual statement, the professional 
reader may have to translate into: clinical experience seems to suggest that such and such 
an observation might be tentatively explained by such and such a working hypothesis, 
requiring a vast amount of further research before the hypothesis can be considered a valid 
theory. If we can from the outset establish this as a mutually accepted convention, I hope 
to avoid being accused of speculative exuberance. 
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Obesity a Disorder 
As a basis for our discussion we postulate that obesity in all its many forms is due to an 
abnormal functioning of some part of the body and that every ounce of abnormally 
accumulated fat is always the result of the same disorder of certain regulatory chanisms. 
Persons suffering from this particular disorder will get fat regardless of whether they eat 
excessively, normally or less than normal. A person who is free of the disorder will never 
get fat, even if he frequently overeats. 
Those in whom the disorder is severe will accumulate fat very rapidly, those in whom it is 
moderate will gradually increase in weight and those in whom it is mild may be able to 
keep their excess weight stationary for long periods. In all these cases a loss of weight 
brought about by dieting, treatments with thyroid, appetite-reducing drugs, laxatives, 
violent exercise, massage, or baths is only temporary and will be rapidly regained as soon 
as the reducing regimen is relaxed. The reason is simply that none of these measures 
corrects the basic disorder. 
While there are great variations in the severity of obesity, we shall consider all the 
different forms in both sexes and at all ages as always being due to the same disorder. 
Variations in form would then be partly a matter of degree, partly an inherited bodily 
constitution and partly the result of a secondary involvement of endocrine glands such as 
the pituitary, the thyroid, the adrenals or the sex glands. On the other hand, we postulate 
that no deficiency of any of these glands can ever directly produce the common disorder 
known as obesity. 
If this reasoning is correct, it follows that a treatment aimed at curing the disorder must be 
equally effective in both sexes, at all ages and in all forms of obesity. Unless this is so, we 
are entitled to harbor grave doubts as to whether a given treatment corrects the underlying 
disorder. Moreover, any claim that the disorder has been corrected must be substantiated 
by the ability of the patient to eat normally of any food he pleases without regaining 
abnormal fat after treatment. Only if these conditions are fulfilled can we legitimately 
speak of curing obesity rather than of reducing weight. 
Our problem thus presents itself as an enquiry into the localization and the nature of the 
disorder which leads to obesity. The history of this enquiry is a long series of high hopes 
and bitter disappointments. 
The History of Obesity 
There was a time, not so long ago, when obesity was considered a sign of health and 
prosperity in man and of beauty, amorousness and fecundity in women. This attitude 
probably dates back to Neolithic times, about 8000 years ago; when for the first time in 
the history of culture, man began to own property, domestic animals, arable land, houses, 
pottery and metal tools. Before that, with the possible exception of some races such as the 
Hottentots, obesity was almost non-existent, as it still is in all wild animals and most 
primitive races. 
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Today obesity is extremely common among all civilized races, because a disposition to the 
disorder can be inherited. Wherever abnormal fat was regarded as an asset, sexual 
selection tended to propagate the trait. It is only in very recent times that manifest obesity 
has lost some of its allure, though the cult of the outsize bust - always a sign of latent 
obesity - shows that the trend still lingers on. 
  
The Significance of Regular Meals 
In the early Neolithic times another change took place which may well account for the fact 
that today nearly all inherited dispositions sooner or later develop into manifest obesity. 
This change was the institution of regular meals. In pre-Neolithic times, man ate only 
when he was hungry and on1y as much as he required too still the pangs of hunger. 
Moreover, much of his food was raw and all of it was unrefined. He roasted his meat, but 
he did not boil it, as he had no pots, and what little he may have grubbed from the Earth 
and picked from the trees, he ate as he went along. 
The whole structure of man's omnivorous digestive tract is, like that of an ape, rat or pig, 
adjusted to the continual nibbling of tidbits. It is not suited to occasional gorging as is, for 
instance, the intestine of the carnivorous cat family. Thus the institution of regular meals, 
particularly of food rendered rapidly, placed a great burden on modern man's ability to 
cope with large quantities of food suddenly pouring into his system from the intestinal 
tract. 
The institution of regular meals meant that man had to eat more than his body required at 
the moment of eating so as to tide him over until the next meal. Food rendered easily 
digestible suddenly flooded his body with nourishment of which he was in no need at the 
moment. Somehow, somewhere this surplus had to be stored. 
Three Kinds of Fat 
In the human body we can distinguish three kinds of fat. The first is the structural fat 
which fills the gaps between various organs, a sort of packing material. Structural fat also 
performs such important functions as bedding the kidneys in soft elastic tissue, protecting 
the coronary arteries and keeping the skin smooth and taut. It also provides the springy 
cushion of hard fat under the bones of the feet, without which we would be unable to 
walk. 
  
The second type of fat is a normal reserve of fuel upon which the body can freely draw 
when the nutritional income from the intestinal tract is insufficient to meet the demand. 
Such normal reserves are localized all over the body. Fat is a substance which packs the 
highest caloric value into the smallest space so that normal reserves of fuel for muscular 
activity and the maintenance of body temperature can be most economically stored in this 
form. Both these types of fat, structural and reserve, are normal, and even if the body 
stocks them to capacity this can never be called obesity. 
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But there is a third type of fat which is entirely abnormal. It is the accumulation of such 
fat, and of such fat only, from which the overweight patient suffers. This abnormal fat is 
also a potential reserve of fuel, but unlike the normal reserves it is not available to the 
body in a nutritional emergency. It is, so to speak, locked away in a fixed deposit and is 
not kept in a current account, as are the normal reserves. 
When an obese patient tries to reduce by starving himself, he will first lose his normal fat 
reserves. When these are exhausted he begins to burn up structural fat, and only as a last 
resort will the body yield its abnormal reserves, though by that time the patient usually 
feels so weak and hungry that the diet is abandoned. It is just for this reason that obese 
patients complain that when they diet they lose the wrong fat. They feel famished and 
tired and their face becomes drawn and haggard, but their belly, hips, thighs and upper 
arms show little improvement. The fat they have come to detest stays on and the fat they 
need to cover their bones gets less and less. Their skin wrinkles and they look old and 
miserable. And that is one of the most frustrating and depressing experiences a human 
being can have. 
Injustice to the Obese 
When then obese patients are accused of cheating, gluttony, lack of will power, greed and 
sexual complexes, the strong become indignant and decide that modern medicine is a 
fraud and its representatives fools, while the weak just give up the struggle in despair. In 
either case the result is the same: a further gain in weight, resignation to an abominable 
fate and the resolution at least to live tolerably the short span allotted to them - a fig for 
doctors and insurance companies. 
Obese patients only feel physically well as long as they are stationary or gaining weight. 
They may feel guilty, owing to the lethargy and indolence always associated with obesity. 
They may feel ashamed of what they have been led to believe is a lack of control. They 
may feel horrified by the appearance of their nude body and the tightness of their clothes. 
But they have a primitive feeling of animal content which turns to misery and suffering as 
soon as they make a resolute attempt to reduce. For this there are sound reasons. 
In the first place, more caloric energy is required to keep a large body at a certain 
temperature than to heat a small body. Secondly the muscular effort of moving a heavy 
body is greater than in the case of a light body. The muscular effort consumes calories 
which must be provided by food. Thus, all other factors being equal, a fat person requires 
more food than a lean one. One might therefore reason that if a fat person eats only the 
additional food his body requires he should be able to keep his weight stationary. Yet 
every physician who has studied obese patients under rigorously controlled conditions 
knows that this is not true. Many obese patients actually gain weight on a diet which is 
calorically deficient for their basic needs. There must thus be some other mechanism at 
work. 
Glandular Theories 
At one time it was thought that this mechanism might be concerned with the sex glands. 
Such a connection was suggested by the fact that many juvenile obese patients show an 
under-development of the sex organs. The middle-age spread in men and the tendency of 
many women to put on weight in the menopause seemed to indicate a causal connection